首页/数据集/Additional file 2 of Hypoxia-inducible APCDD1L-AS1 promotes osimertinib resistance by stabilising DLST to drive tricarboxylic acid cycle in lung adenocarcinoma

Additional file 2 of Hypoxia-inducible APCDD1L-AS1 promotes osimertinib resistance by stabilising DLST to drive tricarboxylic acid cycle in lung adenocarcinoma

Figshare2025-07-09 更新2026-04-28 收录

化疗耐药机制

肿瘤代谢重编程

数据链接：

https://figshare.com/articles/dataset/Additional_file_2_of_Hypoxia-inducible_APCDD1L-AS1_promotes_osimertinib_resistance_by_stabilising_DLST_to_drive_tricarboxylic_acid_cycle_in_lung_adenocarcinoma/29526871 数据链接链接失效反馈

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资源简介：

Supplementary Material 2

应用场景：

创建时间：

2025-07-09

化疗耐药机制

外泌体介导的耐药传递

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DataSheet2_Exogenous proline enhances susceptibility of NSCLC to cisplatin via metabolic reprogramming and PLK1-mediated cell cycle arrest.xlsx

化疗耐药机制

代谢重编程

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结直肠癌脂质代谢

化疗耐药机制

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头颈部鳞状细胞癌基因

化疗耐药机制

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化疗耐药机制

表观遗传调控

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Additional file 2 of Hypoxia-inducible APCDD1L-AS1 promotes osimertinib resistance by stabilising DLST to drive tricarboxylic acid cycle in lung adenocarcinoma

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