LTP expression mediated by autonomous activity of GluN2B-bound CaMKII

NIAID Data Ecosystem2026-05-02 收录

长时程增强机制

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LTP expression mediated by autonomous activity of GluN2B-bound CaMKII raw data

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2024-10-10

hnRNPM deficiency leads to cognitive deficits via disrupting synaptic plasticity

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认知障碍分子机制

RNA metabolism involves complex and regulated processes, some of which include transcription, intracellular transport, translation, and degradation. The involvement of RNA binding proteins in these pr

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STDP机制分析

Summary of stability/plasticity in STDP models.

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Learning rules for two different time scales.

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小脑学习规则

For short-time scale, instantaneous change of postsynaptic [Ca2+]i is α and its average over a long time is β. The difference (α−β) integrated over a short time (∫(α−β)dt) indicates whether the CF act

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Interactome Analysis Reveals Regulator of G Protein Signaling 14 (RGS14) is a Novel Calcium/Calmodulin (Ca 2+ /CaM) and CaM Kinase II (CaMKII) Binding Partner

蛋白质相互作用组学

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Regulator of G Protein Signaling 14 (RGS14) is a complex scaffolding protein that integrates G protein and MAPK signaling pathways. In the adult mouse brain, RGS14 is predominantly expressed in hippoc

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RNA-Seq in hippocampus of control and Jade2-KO mice

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Impairment of synaptic plasticity is involved in a range of pathological conditions such as cognitive deficits. However, how synaptic efficacy is regulated is not fully understood. Here, we report tha

NIAID Data Ecosystem10

LTP expression mediated by autonomous activity of GluN2B-bound CaMKII

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