Interaction effects of knockdown of PML and TNFalpha treatment in HUVEC cells

ChEBI2013-01-10 更新2026-07-21 收录

基因敲降交互效应

细胞炎症反应

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Interaction effects of knockdown of PML and TNFalpha treatment in HUVEC cells

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2013-01-10

Coordinated demethylation of H3K9 and H3K27 is required for rapid inflammatory responses of endothelial cells.

表观遗传调控

细胞炎症反应

Histone H3 lysine-9 di-methylation (H3K9me2) and lysine-27 tri-methylation (H3K27me3) are linked to repression of gene expression, but the functions of repressive histone methylation dynamics during i

NIAID Data Ecosystem80

The role of endothelial MERTK during the inflammatory response in lungs

细胞炎症反应

MERTK信号通路

As a key homeostasis regulator in mammals, the MERTK receptor tyrosine kinase is crucial for efferocytosis, a process that requires remodeling of the cell membrane and adjacent actin cytoskeleton. Mem

NIAID Data Ecosystem60

Differential gene expression profiles by septin 2 suppression and TNF-a treatment in human microvascular endothelial cells

基因表达调控

细胞炎症反应

Arrangements of juntional proteins of endothelial monolayers were altered by both septin 2 KD and TNF-a treatment. Therefore, differential gene expression profiles by septin 2 KD comapred to control a

NIAID Data Ecosystem40

Systematic analysis of the pro and anti-inflammatory activation of mouse myeloid cells

细胞炎症反应

骨髓细胞RNA-seq分析

Inflammation is a fundamental physiological process and a key line of defense against pathogen invasion. Inflammation is exquisitely controlled: Too high and inflammation can be dangerous to the organ

NIAID Data Ecosystem60

Ectopic expression data of bacterial effects on NNE and NPC cells

鼻咽癌分子机制

细胞炎症反应

Compared with nasopharyngeal carcinoma cell line C666-1, we found that pro-inflammatory cytokines in NP69 cells were significantly upregulated after PGN treatment We used microarray technology to iden

NIAID Data Ecosystem50

Interaction effects of knockdown of PML and TNFalpha treatment in HUVEC cells

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