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Homocysteine promotes the pathogenesis of atherosclerosis through circ-PIAS1-5/miR-219a-2-3p/TEAD1 axis

NIAID Data Ecosystem2026-05-02 收录

动脉粥样硬化机制

circRNA与脂质代谢

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1139659 数据链接链接失效反馈

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资源简介：

the biological functions and potential molecular mechanisms of circRNAs in HHcy-induced lipid accumulation leading to atherosclerosis

应用场景：

创建时间：

2024-07-24

动脉粥样硬化机制

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Nicotinic Acid Receptor GPR109A Is Down-Regulated in Human Macrophage-Derived Foam Cells

动脉粥样硬化机制

泡沫细胞

Nicotinic acid (NA) regresses atherosclerosis in human imaging studies and reduces atherosclerosis in mice, mediated by myeloid cells, independent of lipoproteins. Since GPR109A is expressed by human

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Gene expression in primary human macrophages and foam cells. Homo sapiens

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泡沫细胞形成

The aim of the experiment was to determine the effects of 48 hours of treatment with oxidized low density lipoprotein (oxLDL) on gene expression in primary human monocyte-derived macrophages. Overall

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Table_1_Exploration of Crucial Mediators for Carotid Atherosclerosis Pathogenesis Through Integration of Microbiome, Metabolome, and Transcriptome.DOCX

动脉粥样硬化机制

多组学整合分析

BackgroundCarotid atherosclerosis (CAS) is an important cause of stroke. Although interactions between the gut microbiome and metabolome have been widely investigated with respect to the pathogenesis

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LncRNA ICR regulates endothelium inflammation through feedback activation of NF-κB signaling

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血管炎症调控

The responsive and abundant lncRNAs to pro-inflammatory factors in human ECs were screened and validated by RNA sequencing and qPCR. ICAM1-related non-coding RNA (ICR) was identified as the most poten

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Homocysteine promotes the pathogenesis of atherosclerosis through circ-PIAS1-5/miR-219a-2-3p/TEAD1 axis

资源简介：

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