Phl p 1-specific human monoclonal IgE and design of a hypoallergenic group 1 grass pollen allergen fragment.
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The C-terminal domain of PhI p1 was able to inhibit binding of the epitope-specific scFv construct to the same protein fragment. A construct containing the K176A, N179A, and D223A mutation was not able to inhibit antibody binding. The epitiope-specific scFv construct also recognized the C-terminal domain of PhI p1 in ELISA.
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IEDB创建时间：
2013-01-01
