Acute myeloid leukemia doxorubicin-resistant mechanism

NIAID Data Ecosystem2026-05-02 收录

白血病耐药机制

阿霉素耐药

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1131095 数据链接链接失效反馈

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资源简介：

To construct doxorubicin-resistant acute myeloid leukemia cell line and explore the mechanism of drug resistance

创建时间：

2024-07-02

Transcriptome-wide analysis of RNA N6-methyladenosine in Adriamycin-resistant acute myeloid leukemia cells

白血病耐药机制

RNAm6A修饰分析

N6-Methyladenosine (m6A) is the most abundant post-transcriptional modification in eukaryotes, the imbalance of which is reported to be associated with various pathological processes, including drug r

NIAID Data Ecosystem60

Data_Sheet_1_Acute Myeloid Leukemia Chemo-Resistance Is Mediated by E-selectin Receptor CD162 in Bone Marrow Niches.PDF

白血病耐药机制

肿瘤微环境靶向治疗

The interactions of leukemia cells with the bone marrow (BM) microenvironment is critical for disease progression and resistance to treatment. We have recently found that the vascular adhesion molecul

NIAID Data Ecosystem40

Expression profile of shikonin resistant K562 cell line

白血病耐药机制

抗肿瘤药物作用机制

Expression profile of shikonin resistant K562 cell line

ChEBI2011-12-10 更新00

Targeting FLT3-TAZ Signaling to Suppress Drug Resistance in Blast Crisis Chronic Myeloid Leukemia. Targeting FLT3-TAZ Signaling to Suppress Drug Resistance in Blast Crisis Chronic Myeloid Leukemia

白血病耐药机制

FLT3信号通路

Although the development of tyrosine kinase inhibitors (TKIs), including BCR-ABL1 targeted therapies, rendered chronic myeloid leukemia (CML) a manageable condition, acquisition of drug resistance dur

NIAID Data Ecosystem40

Targeting the MTF2-MDM2 Axis Sensitizes Refractory Acute Myeloid Leukemia to Chemotherapy [RNA-Seq]

白血病耐药机制

表观遗传靶向治疗

Deep sequencing has revealed that epigenetic modifiers are the most mutated genes in acute myeloid leukemia (AML). Thus, elucidating epigenetic dysregulation in AML is crucial to understand disease me

NIAID Data Ecosystem10

Acute myeloid leukemia doxorubicin-resistant mechanism

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