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Transcription profiling of human cerebellum from schizophrenia patients and normal subjects

ChEBI2007-06-22 更新2026-07-21 收录

精神分裂症分子机制

小脑转录组

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-4036 数据链接链接失效反馈

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资源简介：

Transcription profiling of human cerebellum from schizophrenia patients and normal subjects

应用场景：

创建时间：

2007-06-22

精神分裂症分子机制

ceRNA网络

Schizophrenia (SCZ) is a serious mental condition with an unknown cause. According to the reports, Brodmann Area 10 (BA10) is linked to the pathology and cortical dysfunction of SCZ, which demonstrate

NIAID Data Ecosystem70

perro-affy-human-186940

精神分裂症分子机制

转基因动物模型

Our laboratory has developed the first mouse model overexpressing a RNA-binding protein, the ELAV-like protein HuD, in the CNS under the control of the CaMKinII alpha promoter. Initial behavioral char

NIAID Data Ecosystem40

Comparing the cerebellar transcriptome of Sacs KO versus WT mice

神经退行性机制

小脑转录组

Autosomal Recessive Spastic Ataxia of Charlevoix-Saguenay (ARSACS) is caused by mutations in SACS gene encoding sacsin. ARSACS patients and mouse models display early degeneration of cerebellum in agr

NIAID Data Ecosystem40

Genome-wide binding profiles of nuclear FGFR1 in Control and Schizophrenia hiPSC-dervied NPC [ChIP-seq]

精神分裂症分子机制

转录调控

We report the genome-wide binding patterns of nuclear FGFR1 in Control and Schizophrenia hiPSC-dervied NPC differntiated in neuronal media for two days. We also report the genome-wide binding pattern

NIAID Data Ecosystem60

Transcriptomic Analysis Shows Decreased Cortical Expression of NR4A1, NR4A2 and RXRB in Schizophrenia and Provides Evidence for Nuclear Receptor Dysregulation

精神分裂症分子机制

核受体转录组学

Many genes are differentially expressed in the cortex of people with schizophrenia, implicating factors that control transcription more generally. Hormone nuclear receptors dimerize to coordinate cont

Figshare2016-12-17 更新40

Transcription profiling of human cerebellum from schizophrenia patients and normal subjects

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