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Proteogenomic analysis of psoriasis reveals discordant and concordant changes in mRNA and protein abundance

ChEBI2015-08-23 更新2026-07-12 收录

银屑病分子机制

蛋白质基因组学

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-67785 数据链接链接失效反馈

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Proteogenomic analysis of psoriasis reveals discordant and concordant changes in mRNA and protein abundance

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2015-08-23

乳腺癌研究

蛋白质基因组学

Additional file 9. Table S2. Differential mutations and SCNA between DTBC and LumA tumors. (A) Significantly (p-value < 0.1) differentially mutated genes with non-synonymous somatic short variants

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c-Jun N-terminal protein kinase 1 (JNK1) is involved in multiple biological processes but its implication in inflammatory skin diseases is still poorly defined. Herein, we studied the role of JNK1 in

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Decrease in Rnase HII and accumulation of lncRNAs/DNA hybrids: a causal implication in psoriasis?

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Long functional non-coding RNAs (lncRNAs) have been in the limelight in aging research because short telomeres are associated with higher levels of TERRA (Telomeric Repeat containing RNA). The genomic

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Table_6_Genetic analysis of potential biomarkers and therapeutic targets in ferroptosis from psoriasis.xlsx

银屑病分子机制

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IntroductionFerroptosis is associated with multiple pathophysiological processes. Inhibition of ferroptosis has received much concern for some diseases. Nonetheless, there is no study comprehensively

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Proteogenomic analysis of psoriasis reveals discordant and concordant changes in mRNA and protein abundance

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