Lactylation of METTL16 promotes cuproptosis via m6A-modification on FDX1 mRNA in gastric cancer. Lactylation of METTL16 promotes cuproptosis via m6A-modification on FDX1 mRNA in gastric cancer
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA932911
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To investigate the targets of METTL16-induced m6A modification, we established AGS lines in which METTL16 has been knocked down by shRNA. Overall design: Comparative gene expression profiling analysis and m6A modification analysis of MeRIP-seq data for AGS control cells (shNC) and its KD derivatives (shMETTL16).
创建时间：
2023-02-08
