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Evidence for CD04+ T Cell Methylation Signatures of Network-oriented SOCS3, ITGAL, and NR4A2 Genes with Hemodynamics in Patients with Pulmonary Arterial Hypertension

NIAID Data Ecosystem2026-03-14 收录

肺动脉高压遗传学

CD4+T细胞甲基化与血流动力学

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE165360 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2023-03-09

肺动脉高压遗传学

基因表达修饰通路

Background: While BMPR2 mutation strongly predisposes to pulmonary arterial hypertension (PAH), only 20% of mutation carriers develop clinical disease. This finding suggests that modifier genes contri

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Altered Smooth Muscle Cell Histone Acetylome and its Regulation in Pulmonary Hypertension

肺动脉高压遗传学

平滑肌细胞乙酰化修饰

Cleavage Under Targets and Release Using Nuclease (CUT&RUN) was performed using CUTANA protocol v1.6 [www.epicypher.com]. Human pulmonary arterial smooth muscle cells (hPASMCs) were treated with vehic

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DataSheet1_Identification of novel genetic variants, including PIM1 and LINC01491, with ICD-10 based diagnosis of pulmonary arterial hypertension in the UK Biobank cohort.docx

肺动脉高压遗传学

药物靶点预测

Pulmonary arterial hypertension (PAH) is characterized by remodeling and narrowing of the pulmonary vasculature which results in elevations of pulmonary arterial pressures. Here, we conducted a genome

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Additional file 2: of Rare variants in SOX17 are associated with pulmonary arterial hypertension with congenital heart disease

肺动脉高压遗传学

先天性心脏病基因变异

Table S4. Variants in known CHD risk genes. (XLSX 46 kb)

Figshare2018-07-21 更新40

Evidence for CD04+ T Cell Methylation Signatures of Network-oriented SOCS3, ITGAL, and NR4A2 Genes with Hemodynamics in Patients with Pulmonary Arterial Hypertension (Array2 DataSet). Evidence for CD04+ T Cell Methylation Signatures of Network-oriented SOCS3, ITGAL, and NR4A2 Genes with Hemodynamics in Patients with Pulmonary Arterial Hypertension (Array2 DataSet)

肺动脉高压遗传学

甲基化生物标志物

We hypothesized that methylation dynamics in circulating CD04+ T cells may underlie a precise transcriptional reprogramming that associates with the cardiopulmonary hemodynamic profile of affected pat

NIAID Data Ecosystem30

Evidence for CD04+ T Cell Methylation Signatures of Network-oriented SOCS3, ITGAL, and NR4A2 Genes with Hemodynamics in Patients with Pulmonary Arterial Hypertension

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