IntroductionSepsis is characterized by life-threatening organ dysfunction caused by dysregulated host response to infection. A key contributor is the disruption of neutrophil-endothelial interactions.
Among other effects, post-translational modifications (PTMs) have been shown to exert their function via the modulation of protein-protein interactions. For twelve different main PTM-types and associa
This network was assembled automatically by INDRA (http://indra.bio) by processing all available biomedical literature with multiple machine reading systems, and integrating curated pathway databases.
