首页/数据集/Genes activated by 5-aza-dC (DAC) in pancreatic cancer (KPC-Brca1) epithelial cells

Genes activated by 5-aza-dC (DAC) in pancreatic cancer (KPC-Brca1) epithelial cells

ChEBI2013-01-01 更新2026-07-21 收录

胰腺癌表观遗传学

药物激活基因分析

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-42365 数据链接链接失效反馈

官方服务：

资源简介：

Genes activated by 5-aza-dC (DAC) in pancreatic cancer (KPC-Brca1) epithelial cells

应用场景：

创建时间：

2013-01-01

胰腺癌表观遗传学

染色质可及性分析

Damaged tissues have increased risk of cancer development through poorly understood mechanisms. In the pancreas, tissue damage collaborates with activating mutations in the Kras oncogene to dramatical

NIAID Data Ecosystem110

ChIP-seq of PDAC tissues grown as patient-derived tumor xenografts

胰腺癌表观遗传学

PDX模型ChIP-seq分析

ChIP-seq of PDAC tissues grown as patient-derived tumor xenografts

ChEBI2018-05-29 更新50

Development of LHX6-inducible system in PK-1 and PK-9 cells. Development of LHX6-inducible system in PK-1 and PK-9 cells

胰腺癌表观遗传学

基因功能验证

Epigenetic gene silencing by aberrant DNA methylation leads to loss of key cellular pathways in tumorigenesis. DNA methylation-mediated silenced genes in pancreatic cancer were searched by methyl-CpG

NIAID Data Ecosystem40

SIRT6 suppresses pancreatic cancer through control of Lin28b [miRNA-seq]

胰腺癌表观遗传学

microRNA靶点分析

Chromatin remodeling proteins are frequently dysregulated in human cancer, yet little is known about how they control tumorigenesis. Here, we uncover an epigenetic program mediated by the NAD+-depende

NIAID Data Ecosystem50

Cell differentiation grade determines distinct FOXA2 contributions to the cis-regulatory networks of pancreatic cancer cells [ATAC-seq]

胰腺癌表观遗传学

癌细胞调控

Abnormal differentiation contributes to the spectrum of aberrant properties of tumor cells. Differentiation of both normal and tumor cells is controlled by regulatory networks enforced by transcriptio

NIAID Data Ecosystem40

Genes activated by 5-aza-dC (DAC) in pancreatic cancer (KPC-Brca1) epithelial cells

资源简介：

相关数据集