Recurrent deletions in clonal hematopoiesis are driven by Microhomology-mediated end joining
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA707245
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资源简介：
In this work we demonstrate that the most common recurrent deletions in clonal hematopoiesis are the result of PARP1 dependent repair of DSBs by a sub-pathway of the MMEJ that is POLQ independent.
创建时间：
2021-03-07
