Metabolites linked to changes in choline kinase-α (CK-α) expression and drug resistance, which contribute to survival and autophagy mechanisms, are attractive targets for breast cancer therapies. We p
Purpose: To assess if NLK modulates ER target gene expression, we performed transcriptome sequencing following NLK inhibition or tamoxifen treatment in the BT483 or T47D-TamR cells under estrogen-depr
Survivin (BIRC5) mRNA expression data were retrieved from a gene-expression profiling dataset (202094_x from Kaplan–Meier Plot database) of 2046 patients with ERα-positive breast cancer and 928 patien
The differential expressed genes of estrogen receptor positive (ER+) breast cancer cell line MCF-7 and its endocrine resistant cell lines: tamoxifen resistant (TAMR) and long-term estrogen deprivation
