Results support the important role of both TOP2α/170 and TOP2α/90 as determinants of sensitivity/resistance to TOP2α-targeting agents. Overall design: CRISPR/Cas9 with homology-directed repair was uti
Annotated sequence files in .ape format (comparable with Genbank) of knock-ins generated by CRISPR/Cas9-mediated genome editing. Unmodified genomic sequence is also provided.
FASTQs from amplicon sequencing of cDNA and gDNA after CRISPR editing of putative regulatory and control variants. Files are contained in tarballs grouped by sequencing run. 20190312 is a rerun of the
RNA-seq on HepG2 cells treated with a CRISPR gRNA against MAK16. (MAK16-BGHcLV12) For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/R
