Table S1-S12 from Pathways Impacted by Genomic Alterations in Pulmonary Carcinoid Tumors

Table S1: Clinicopathologic features Table S2: Statistical analysis of correlation between number of mutations with sevaral clinical characteristics Table S3: List of copy number variations in different types of pulmonary neuroendocrine tumors as reported by different studies. Lung typical carcinoid (TC), Lung atypical carcinoid Table S4: Table S2. List of genes differentially expressed genes in regions of corresponding copy number variation (red signifies amplification and blue represent deletion) Table S5: Functional analysis using the DAVID functional annotation database Table S6: A table of significantly mutated genes determine by MutSigCV analysis. Table S7: Ingenuity pathway analysis of Significant mutated gene with q<0.5 Table S8: MutSig analysis of chromatin remodeling genes Table S9: A listed of validated cancer causal genes by ingenuity variant analysis and Oncotator annotation and filtering Table S10. Common CNV genes identified by both SNP array and WGS Table S11: Canonical pathways identified by Ingenuity Pathway analysis of Fernandez-Cuesta et al RNASeq data Table S12: List of chromatin remodelling genes with damaging and functional variants by analysis pipeline