Loss-of-function mutations of p16(INK4a) have been identified in a large number of human tumors. An established biochemical function of p16 is its ability to specifically inhibit cyclin D-dependent ki
One hundred and six miRNA were significantly modulated. Of these, 66 were up regulated and 40 were down regulated. Values are given as Log10 of the fold change (2−(mean ΔΔCt)).Significantly modulated
p-S1, p-S2, p-S3, p-S4, p-S5 and p-S6 are short for p-values of each step, from step 1 to step 6. Step 1, the association between the phenotypes of endoxifen sensitivity and ∼13,000 gene/transcript cl
