BCR-ABL1 promotes leukemia by converting p27 into a cytoplasmic oncoprotein. Mus musculus
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA254523
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资源简介：
Coordinated BCR-ABL1 kinase-dependent and -independent mechanisms convert p27 from a nuclear tumor suppressor to a cytoplasmic oncogene. Persistence of oncogenic p27 functions despite effective inhibition of BCR-ABL1 may contribute to resistance to tyrosine kinase inhibitors. Overall design: BCR-ABL1 induced p27 versus knockout, controlling with Empty vector p27 versus knock out
创建时间：
2014-07-07
