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资源简介：
ALI/ARDS acute lung injury/acute respiratory distress syndrome.
应用场景：
创建时间：
2014-02-27
相关数据集
Cellular stress modulates severity of the inflammatory response in lungs via cell surface BiP
As BiP is involved in the ARDS mechanism of disease, we analyzed the proteomic profile of lung tissue challenged with LPS and/or treated with 4-PBA to identify pathways and components that link BiP an
NIAID Data Ecosystem60
DataSheet1_Cinobufagin alleviates lipopolysaccharide-induced acute lung injury by regulating autophagy through activation of the p53/mTOR pathway.ZIP
Acute lung injury (ALI) is a severe clinical disorder characterized by dysregulated inflammatory responses, leading to high rates of morbidity and mortality. Cinobufagin, a primary component isolated
NIAID Data Ecosystem10
Table 1_Extracellular vesicles from mesenchymal stromal cells as a promising therapy for ARDS: a systematic review of preclinical studies.docx
IntroductionMesenchymal stromal cell-derived extracellular vesicles (MSC-EVs) have emerged as a promising cell-free therapeutic strategy for acute respiratory distress syndrome (ARDS), a condition wit
NIAID Data Ecosystem00
Antibiotic-induced microbiome depletion ameliorates LPS-induced acute lung injury in mice.
In this research, treatment with ABX significantly attenuated LPS-induced lung injury of mice. Considering the key role of gut microbiota in immune system, it is likely that alternations in the gut mi
NIAID Data Ecosystem50
Gut-Lung Axis Modulation by Hydroxysafflor Yellow A Mitigates Acute Lung Injury
Hydroxysafflor yellow A (HSYA) is the main active ingredient of safflower, a medicinal plant that has beneficial effects on a variety of lung diseases. However, the impact of HSYA on acute lung injury
NIAID Data Ecosystem10
