Additional file 1 of Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes
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https://springernature.figshare.com/articles/Additional_file_1_of_Amorphous_SiO2_nanoparticles_promote_cardiac_dysfunction_via_the_opening_of_the_mitochondrial_permeability_transition_pore_in_rat_heart_and_human_cardiomyocytes/12270122
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Additional file 1: Supplementary Figure 1. Perfusion for 30 min with nanoSiO2 did not cause structural alterations or inflammation in the heart apex. H&E staining of: A) untreated, B) 40 μg/mL nanoSiO2 perfusion, and C) 200 μg/mL nanoSiO2 perfusion. Supplementary Figure 2. nanoSiO2 accumulates in mitochondria from ventricle myocytes. Representative TEM micrograph of mitochondria showing swelling and its assessment by TEM-EDS from: (A) untreated rat CMs, (B) nanoSiO2 exposed rat CMs. (C) Quantification of Si content from EDS spectra.
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figshare
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2020-05-08
