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FZD7 Plays a Critical Role in Triple Negative Breast Cancer Proliferation
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创建时间：
2025-01-24
相关数据集
Genomic Regulation of Invasion by STAT3 in Triple Negative Breast Cancer
Breast cancer is a heterogeneous disease comprised of four molecular subtypes defined by whether the tumor-originating cells are luminal or basal epithelial cells. Breast cancers arising from the lumi
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SMARCA4 loss-of-function induces cisplatin resistance to activate YAP1-dependent epithelial-to-mesenchymal in triple negative breast cancer
We report transcriptome profiling (RNA-seq) for SMARA4 knock-out to elucidate the association with cisplatin-resistance and SMARCA4 loss-of-function using triple negative breast cancer cell lines. Ove
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Table S11 from Basal-A Triple-Negative Breast Cancer Cells Selectively Rely on RNA Splicing for Survival
Table S11: Differentially expressed transcripts in E7107- vs DMSO-treated MB468 cells
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ChIP-seq of E4F1 in SUM159 triple negative breast cancer cell line
E4F1 is a ubiquitously expressed zinc-finger protein of the Gli-Kruppel family that was first identified, more than 30 years ago, as a cellular target of the adenoviral oncoprotein E1A13S (Ad type V),
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Regulation of cellular heterogeneity and symmetric/asymmetric divisions in triple-negative breast cancer
Differentiation events contribute to cellular heterogeneity within tumors and influence disease progression and response to therapy. Here we dissect mechanisms controlling intratumoral heterogeneity w
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