Caco-2 cells were infected with influenza virus (PR8) at different MOI (0.2, 0.4 and 0.8) for 24 hrs. Cells were then infected with E. coli LF82, LF82 ΔfimH or S15 (MOI 25) for 2 hrs. As controls, cel
To investigate viral-bacterial synergy in the allergic host, we developed a 'triple-threat model' by combining our fungal asthma model with a well-established model of IAV and Spn co-infection.
Influenza A virus (IAV) predisposes individuals to secondary infections with the bacterium Streptococcus pneumoniae (the pneumococcus). Infections may manifest as pneumonia, sepsis, meningitis or otit
In superinfection studies, two Staphylococcus aureus V8 strains administered to mice, post non-lethal dose injection with influenza virus, resulted significantly different mice lethality. The ATCC str