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Transcription profiling of rat Fcarcinoma cells during FGF-1 induced Epithelial-mesenchymal transition

ChEBI2008-04-07 更新2026-07-21 收录

癌症转移机制

上皮-间质转化

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-5664 数据链接链接失效反馈

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Transcription profiling of rat Fcarcinoma cells during FGF-1 induced Epithelial-mesenchymal transition

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创建时间：

2008-04-07

乳腺癌

上皮-间质转化

Additional file 2. Turquoise (EMT) subnetwork. The list of genes, module membership, the logarithm of fold change, differential analysis. adj-p.value, and gene-significance statistics.

DataCite Commons2021-11-21 更新90

NFIC1 suppresses migration and invasion of breast cancer cells through interferon-mediated Jak-STAT pathway

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Jak-STAT信号通路

NFIC1, the longest isoform of NFIC, is essential for the regulation on spatiotemporal expressions of drug-metabolizing genes in liver. However, the role of NFIC1 in breast cancer is not clear. Here we

NIAID Data Ecosystem40

shRNAi profiling of human MDA-MB-231 cells expressing anti-p53 (shp53) short-hairpin RNAs treated with TFGbeta or control

癌症转移机制

转录组功能分析

TGFβ ligands act as tumor suppressors in early stage tumors but are paradoxically diverted into potent prometastatic factors in advanced cancers. The molecular nature of this switch remains enigmatic.

NIAID Data Ecosystem30

Additional file 2 of An integrated multi-omics approach to identify regulatory mechanisms in cancer metastatic processes

癌症转移机制

多组学整合分析

Additional file 2. Gene set characterization. Gene set characterization of the DE genes between early and late stages. The downregulated genes are significantly enriched for cancer-related gene signat

NIAID Data Ecosystem50

Flag-RBM47 HITS-CLIP in brain metastatic derivatives of MDA-MB-231

RNA结合蛋白靶标

癌症转移机制

Brain metastatic breast cancer cells were subjected to HITS-CLIP to identify the targets of the RNA binding protein RBM47 MDA231-BrM2a is characterized in PMID: 19421193; Related data published toget

NIAID Data Ecosystem40

Transcription profiling of rat Fcarcinoma cells during FGF-1 induced Epithelial-mesenchymal transition

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