The effect of YAP/TAZ knockdown on the intestinal epithelium
收藏官方服务:
资源简介:
The effect of YAP/TAZ knockdown on the intestinal epithelium
应用场景:
创建时间:
2014-07-18
相关数据集
Yap dependent reprogramming of Lgr5+ stem cells drives intestinal regeneration and cancer. Mus musculus
Hippo signalling has been implicated as a key regulator of tissue regeneration. In the intestine, ex vivo organoid cultures model aspects of crypt epithelial regeneration. Therefore in order to uncove
NIAID Data Ecosystem70
Transcriptional effects of YAP/TAZ knockdown in fibroblasts. Transcriptional effects of YAP/TAZ knockdown in fibroblasts
Gene expression profiles of the cells composing the dorsal skin of young control and YAP/TAZ knockout mice with the skin of a old mouse. Overall design: Single cell profiles were derived from the dors
NIAID Data Ecosystem40
Loss of Hepatocyte-Nuclear-Factor-1α Impacts on Adult Mouse Intestinal Epithelial Cell Growth and Cell Lineages Differentiation. Mus musculus
Although hepatocyte-nuclear-factor-1α (Hnf1α) is crucial for pancreas and liver functions, it is believed to play a limited functional role for intestinal epithelial functions. The aim of this study w
NIAID Data Ecosystem30
Differential Gene expression in the mouse mammary tumors of PyMT-Malat1 wild-type (WT), PyMT-Malat1 knockout (KO) and PyMT-Malat1 knockout with Malat1 transgene expression (TG)
Previously, lncRNA Malat1 knockout mice were generated by insertional inactivation. By crossing this line to MMTV-PyMT mammary tumor mouse model, we produced PyMT;Malat1 wild-type (WT) and PyMT;Malat1
NIAID Data Ecosystem40
YAP and TAZ are transcriptional co-activators of AP-1 proteins and STAT3 during breast cellular transformation (ChIP-seq)
The YAP and TAZ paralogues, the ultimate effectors of the Hippo signaling pathway, are deregulated in many cancer types. They are transcriptional co-activators that are recruited to their target sit
NIAID Data Ecosystem40



