TFE3 fusions escape from controlling of mTOR signaling pathway and accumulate in the nucleus promoting genes expression in Xp11.2 translocation renal cell carcinomas.
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https://www.ncbi.nlm.nih.gov/sra/SRP507047
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资源简介：
ChIP analysis demonstrated that TFE3 fusions could bind to the promoters of the target genes as a wild-type TFE3 protein. Overall design: Chip-seq for TFE3 in UOK120 cells and UOK109 cells
创建时间：
2024-08-15
