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Loss of p21 expression enhances DNA damage, cholestasis and hepatocarcinogenesis in the liver

ChEBI2015-03-01 更新2026-07-21 收录

肝细胞癌机制

肝脏分子病理学

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-61100 数据链接链接失效反馈

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Loss of p21 expression enhances DNA damage, cholestasis and hepatocarcinogenesis in the liver

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创建时间：

2015-03-01

辐射转录组学

肝细胞癌机制

Background: mRNA interactions with each other and other signaling molecules define different biological pathways and functions. Researchers have been investigating various tools to analyze these types

NIAID Data Ecosystem80

NLRP12 WT and KO mice fecal samples Raw 16S rRNA sequence reads. NLRP12 WT and KO mice fecal samples

消化道微生物组

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NLRP12 suppresses hepatocellular carcinoma via downregulation of cJun N-terminal kinase activation in the hepatocyte

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snRNAseq of bCat-Kras tumors in normal and NASH diet

肝细胞癌机制

Hippo信号通路调控

Current models of hepatocellular tumorigenesis propose that the chronic injury and inflammation in NASH are mutagenic and growth promoting which can eventually lead to HCC. However, contrary to this m

NIAID Data Ecosystem50

Tid1 Deficiency Disrupting Mitochondria to Drive NASH-Dependent Hepatocellular Carcinoma Progression. Tid1 Deficiency Disrupting Mitochondria to Drive NASH-Dependent Hepatocellular Carcinoma Progression

肝细胞癌机制

HCC肝细胞癌

Non-alcoholic fatty liver disease/steatohepatitis (NAFLD/NASH) is a significant risk factor for hepatocellular carcinoma (HCC). However, a preclinical model of progressive NAFLD/NASH is largely lackin

NIAID Data Ecosystem70

3D organization profiling of human hepatocellular carcinoma cell line PLC/PRF/5 in comparison with normal human liver cell line L02 by in situ Hi-C

三维基因组学

肝细胞癌机制

Genetic and epigenetic alterations accumulate in the process of hepatocellular carcinogenesis, but the role of genomic spatial organization in HCC is still unknown. Overall design: We performed in sit

NIAID Data Ecosystem60

Loss of p21 expression enhances DNA damage, cholestasis and hepatocarcinogenesis in the liver

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