MiR-155 promotes blood-brain barrier dysfunction in neuroinflammation

NIAID Data Ecosystem2026-03-11 收录

血脑屏障功能障碍

炎症分子机制

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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE44694 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2019-10-03

miRNA array for LPS-activated tnc+/+ and tnc-/- macrophages

巨噬细胞免疫调节

炎症分子机制

Endogenous molecules generated upon pathogen invasion or tissue damage serve as danger signals that activate host defence, however their precise immunological role remains unclear. Tenascin-C is an ex

NIAID Data Ecosystem50

The Cerebral Microvasculature in Schizophrenia: A Laser Capture Microdissection Study

精神分裂症脑微血管转录组

血脑屏障功能障碍

BackgroundPrevious studies of brain and peripheral tissues in schizophrenia patients have indicated impaired energy supply to the brain. A number of studies have also demonstrated dysfunction of the m

NIAID Data Ecosystem40

Additional file 5 of Minocycline reduces inflammatory response and cell death in a S100B retina degeneration model

视网膜疾病蛋白质组学

炎症分子机制

Additional file 5: Supplementary table 5. Functional annotation analysis of the differentially abundant proteins.

Figshare2020-12-14 更新00

Human lymph node derived fibroblastic and double negative reticular cells alter their chemokines and cytokine expression profile following inflammatory stimuli

免疫细胞功能

炎症分子机制

Lymph node is a secondary lymphoid organ that has structural organization related to their immune function, which is highly ordered and compartmentalized. Lymph nodes shelter immune cells and also fib

NIAID Data Ecosystem40

Mosaic deletion of claudin-5 reveals rapid non-cell-autonomous consequences of blood-brain barrier leakage

血脑屏障功能障碍

单细胞转录组学

Claudin-5 (CLDN5) is an endothelial tight junction protein essential for blood-brain barrier (BBB) formation. Abnormal CLDN5 expression is common in brain disease, and knockdown of Cldn5 at the BBB ha

NIAID Data Ecosystem10

MiR-155 promotes blood-brain barrier dysfunction in neuroinflammation

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