首页/数据集/Epigenetic and immune function profiles associated with post-traumatic stress disorder

Epigenetic and immune function profiles associated with post-traumatic stress disorder

ChEBI2010-05-12 更新2026-07-21 收录

创伤后应激障碍生物标志物

表观遗传与免疫关联分析

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-21282 数据链接链接失效反馈

官方服务：

资源简介：

Epigenetic and immune function profiles associated with post-traumatic stress disorder

应用场景：

创建时间：

2010-05-12

相关数据集

PTSD Normalized data

创伤后应激障碍生物标志物

时序基因表达分析

PTSD - Posttraumatic stress disorder. 33 samples taken from PMBCs of survivors of psychological trauma, in two time points: in ER, few hours after the truma, and four months later. Some of the patient

NIAID Data Ecosystem30

Gene Networks Specific for Innate Immunity Define Post-traumatic Stress Disorder [RNA-Seq]

创伤后应激障碍生物标志物

先天免疫基因

The molecular factors involved in the development of Post-Traumatic Stress Disorder (PTSD) remain poorly understood. Previous transcriptomic studies investigating the mechanisms of PTSD apply targeted

NIAID Data Ecosystem30

Prediction of Possible Biomarkers and Novel Pathways Conferring Risk to Post-Traumatic Stress Disorder

创伤后应激障碍生物标志物

基因功能富集分析

Post-traumatic stress disorder is one of the common mental ailments that is triggered by exposure to traumatic events. Till date, the molecular factors conferring risk to the development of PTSD is no

Figshare2016-12-21 更新20

Epigenomic study of Post-traumatic Stress Disorder among OIF/OEF veterans. Homo sapiens

创伤后应激障碍生物标志物

表观遗传学分析

Emerging knowledge suggests that post-traumatic stress disorder (PTSD) is causally associated with epigenetic changes although its molecular underpinnings are still largely elusive. We postulate that

NIAID Data Ecosystem30

Pilot study of the association between microbiome characteristics and the development of adverse posttraumatic neuropsychiatric sequelae after traumatic stress exposure

创伤后应激障碍生物标志物

微生物组

Background: Patients suffering trauma often experience high rates of post-traumatic neuropsychiatric sequelae (APNS). The biologic mechanisms promoting APNS are currently unknown, but the microbiota-g

NIAID Data Ecosystem20