官方服务:
资源简介:
To investigate the potential role of PARP1 inhibition in improving cardiac function after myocardial infarction in mice by reducing macrophage polarization.
应用场景:
创建时间:
2024-08-01
相关数据集
DataSheet5_Three-Dimensionally Printed Ti2448 With Low Stiffness Enhanced Angiogenesis and Osteogenesis by Regulating Macrophage Polarization via Piezo1/YAP Signaling Axis.zip
Previous studies have found that the novel low-elastic-modulus Ti2448 alloy can significantly reduce stress shielding and contribute to better bone repair than the conventional Ti6Al4V alloy. In this
NIAID Data Ecosystem20
Cardiac conduction system regeneration prevents arrhythmias after myocardial infarction
Arrhythmias are a hallmark of myocardial infarction (MI) and increase patient mortality. The cardiac conduction system is implicated in arrhythmias, but how it is altered following MI is poorly unders
NIAID Data Ecosystem10
Pharmacological inhibition of the glucocorticoid regenerating enzyme 11ßhydroxysteroid dehydrogenase type 1 after myocardial infarction preserves cardiac function in a translational pig model
Background and Purpose: Adrenal glucocorticoids (GC) increase acutely after myocardial infarction (MI) and thereafter tissue levels are maintained selectively within cells expressing the enzyme 11ßHyd
NIAID Data Ecosystem00
Baseline Characteristics of Patients Admitted for Acute Myocardial Infarction, Stratified by Patient Reporting of Financial Stress.
Note: SD = Standard Deviation; BMI = Body Mass Index; CAD = Coronary Artery Disease; CABG = Coronary Artery Bypass Graft; PCI = Percutaneous Coronary Intervention; AMI = Acute Myocardial Infarction; S
NIAID Data Ecosystem20
datasheet1_4-Hydroxyisoleucine Alleviates Macrophage-Related Chronic Inflammation and Metabolic Syndrome in Mice Fed a High-Fat Diet.docx
In obesity, macrophages and other immune cells accumulate in organs affected by insulin, leading to chronic inflammation and insulin resistance. 4-Hydroxyisoleucine (4-HIL) is a non-protein amino acid
NIAID Data Ecosystem10



