首页/数据集/siRNA knockdown of ribosomal protein gene RPL19 abrogates the aggressive phenotype of human prostate cancer

siRNA knockdown of ribosomal protein gene RPL19 abrogates the aggressive phenotype of human prostate cancer

ChEBI2011-12-08 更新2026-07-21 收录

前列腺癌分子机制

基因功能实验

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-26774 数据链接链接失效反馈

官方服务：

资源简介：

siRNA knockdown of ribosomal protein gene RPL19 abrogates the aggressive phenotype of human prostate cancer

应用场景：

创建时间：

2011-12-08

前列腺癌分子机制

基因表达调控

The goals of this study is to find effect of overexpression-TFEB in prostate cancer cell (DU145 cell line). Overall design: To further explore the potential molecular mechanisms of how TFEN affects ca

NIAID Data Ecosystem80

Constitutively Active Androgen Receptor Variants Upregulate Expression of Mesenchymal Markers in Prostate Cancer Cells

前列腺癌分子机制

激素受体功能

Androgen receptor (AR) signaling pathway remains the foremost target of novel therapeutics for castration-resistant prostate cancer (CRPC). However, the expression of constitutively active AR variants

NIAID Data Ecosystem60

SMAD3 promotes the expression and activity of androgen receptor in prostate cancer [RNA-seq]

前列腺癌分子机制

转录调控

Overexpression of androgen receptor (AR) is the primary cause of castration-resistant prostate cancer, although mechanisms upregulating AR transcription in this context are not well understood. Our RN

NIAID Data Ecosystem10

Examining alterations in gene expression of PC3-wt (PSMA -ve) vs PC3-PSMA (PSMA +ve) cells

前列腺癌分子机制

基因表达谱分析

The biological role of PSMA, a biomarker expressed in prostate cancer and by the neovasculature of solid tumors, remains unknown. Although PSMA processes folates in our duodenum, and in the brain clea

NIAID Data Ecosystem70

Table S1 from SOX4-Mediated Post-transcriptional Suppression of PTEN via miR-106b∼25 Cluster Contributes to Prostate Cancer Aggressiveness

前列腺癌分子机制

转录后调控

Table S1 shows the datasets used in this study.

NIAID Data Ecosystem30