Additional file 2: of Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite
Quantitative real-time PCR validations of the FPKM-based expression of unigenes by RNA-seq. Note: RE indicates relative expression levels of unigenes by qRT-PCR. (RAR 18 kb)
