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Table S4 from Ontogeny Dictates Oncogenic Potential, Lineage Hierarchy, and Therapy Response in Pediatric Leukemia

Figshare2026-03-02 更新2026-04-28 收录

白血病分子机制

祖细胞基因表达

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https://figshare.com/articles/dataset/Table_S4_from_Ontogeny_Dictates_Oncogenic_Potential_Lineage_Hierarchy_and_Therapy_Response_in_Pediatric_Leukemia/31443329 数据链接链接失效反馈

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Differentially expressed genes in LMPP Early GMPs

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2026-03-02

白血病分子机制

肿瘤抑制基因

Lentiviral constructs expressing candidate tumoure suppressor genes were transduced into 697 cells. Of the candidates FOXO3, two isoforms of PRDM1 (⍺ and β) suppressed growth in-vitro and in-vivo. To

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ZFP36L2 post-transcriptionally controls acute myeloid leukemia differentiation (MOLM13 HiC). ZFP36L2 post-transcriptionally controls acute myeloid leukemia differentiation (MOLM13 HiC)

白血病分子机制

三维基因组学

Genome-wide assessment of TADs in AML cells Overall design: TAD domains in MOLM13 AML cells

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Interaction of c-Myb with p300 is required for the induction of acute myeloid leukemia (AML) by human AML oncogenes

白血病分子机制

致癌蛋白相互作用

Interaction of c-Myb with p300 is required for the induction of acute myeloid leukemia (AML) by human AML oncogenes

ChEBI2012-12-31 更新00

Gene expression profile of the human T-ALL cell line JURKAT after TYK2 and STAT1 knockdown

白血病分子机制

基因靶向治疗

Targeted molecular therapy has yielded remarkable outcomes in certain cancers, but specific therapeutic targets remain elusive for many others. As a result of two independent RNA interference (RNAi) s

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Effects of Vhl deletion in Dmp1-expressing cells alterering gene expression of megakaryocyte-erythroid progenitor cells (MEPs). Effects of Vhl deletion in Dmp1-expressing cells alterering gene expression of megakaryocyte-erythroid progenitor cells (MEPs)

祖细胞基因表达

髓外造血调控

In recent years, general hypoxia-inducible factor (HIF)-prolyl hydroxylase (PHD) enzyme inhibitors have been developed for the treatment of anemia due to renal disease and osteoporosis. However, it re

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Table S4 from Ontogeny Dictates Oncogenic Potential, Lineage Hierarchy, and Therapy Response in Pediatric Leukemia

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