H3K4me1 marks DNA regions hypomethylated during aging in human stem and differentiated cells
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2019-04-04
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A hyper-quiescent chromatin state formed during aging is reversed by regeneration
Epigenetic alterations are a key hallmark of aging but have not been extensively explored in tissues. Here, using naturally aged murine liver as a model and extending study to other quiescent tissues,
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Additional file 2: of Screening for genes that accelerate the epigenetic aging clock in humans reveals a role for the H3K36 methyltransferase NSD1
Information for the samples with developmental disorders (cases) that were included in the main screen (Nâ =â 367). (TSV 216 kb)
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Mapping H4K20me3 onto the chromatin landscape of senescent cells indicates a function in control of cell senescence and tumor suppression through preservation of genetic and epigenetic stability.
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