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Cooperative Signaling Through the STAT3 and NF-kB Pathways in Subtypes of DLBCL

ChEBI2010-05-27 更新2026-07-21 收录

淋巴瘤信号通路

肿瘤分子机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-10009 数据链接链接失效反馈

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Cooperative Signaling Through the STAT3 and NF-kB Pathways in Subtypes of DLBCL

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创建时间：

2010-05-27

肿瘤分子机制

自噬信号通路

HMGB1 is important for tumor development of autophagy-deficient liver. However, detailed molecular signals impacted by HMGB1 is unclear. RNA seq data will provide a comprehensive gene expression profi

NIAID Data Ecosystem90

File S1 - MMP9 Processing of HSPB1 Regulates Tumor Progression

肿瘤分子机制

蛋白质功能调控

Contains the following: Figure S1. The effect of soluble HSPB1 on tumor transendothelial migration A. Transendothelial migration of HOS cells in the presence of HSPB1 or an HSPB1-neutralizing antibody

NIAID Data Ecosystem40

Table_1_miR-484: A Potential Biomarker in Health and Disease.docx

微小RNA

肿瘤分子机制

Disorders of miR-484 expression are observed in cancer, different diseases or pathological states. There is accumulating evidence that miR-484 plays an essential role in the development as well as the

NIAID Data Ecosystem30

Supplementary Materials

肿瘤分子机制

甲状腺癌基因组学

Table S1: HARRIS_HYPOXIA gene set and REACTOME_AUTOPHAGY gene set; Table S2: Hypoxia phenotype score and autophagy phenotype score of each PTC sample in the TCGA-THCA dataset; Table S3: The qPCR prime

Zenodo2025-04-07 更新70

Additional file 5: of Thymic Epithelial Tumors phenotype relies on miR-145-5p epigenetic regulation

肿瘤分子机制

miRNA调控

Putative targets of the best 9 of 22 miRNAs common to histotype groups of thymic tumors compared to normal tissue (Ganci et al., [43]) selected by target prediction analysis of genes deregulated in Th

Figshare2017-05-10 更新40

Cooperative Signaling Through the STAT3 and NF-kB Pathways in Subtypes of DLBCL

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