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Identification of Metastasis-prone Lung Adenocarcinoma Cell Population That Is Sensitive to Notch Inhibition

ChEBI2011-02-24 更新2026-07-21 收录

肺腺癌转移机制

信号通路靶向治疗

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-15587 数据链接链接失效反馈

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Identification of Metastasis-prone Lung Adenocarcinoma Cell Population That Is Sensitive to Notch Inhibition

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创建时间：

2011-02-24

肿瘤耐药机制

信号通路靶向治疗

Taxanes are widely used in chemotherapy, but intrinsic and acquired resistance limit the clinical outcomes. Studies showed tumor interaction with suppressive macrophages plays a key role in taxane res

NIAID Data Ecosystem80

Differential expression analysis of primary AML cells treated with AC220 vs DMSO in endothelial cell co-culture.

化疗耐药机制

信号通路靶向治疗

Although chemotherapy can successfully induce remission in FLT3/ITD positive acute myeloid leukemia (AML) patients, many, especially those with a high ratio of the FLT3/ITD versus wild type allele (FL

NIAID Data Ecosystem50

Targeting Notch signaling to restore neural development and behavior in mouse models of ASD [RNAseq_htr]

发育疾病机制

信号通路靶向治疗

Autism spectrum disorder (ASD) is a neurodevelopmental disorder with diverse genetic and environmental origins, yet whether these factors converge on common molecular pathways remains unclear. This st

NIAID Data Ecosystem40

Supplementary Material for: Lysophosphatidic Acid Induces Ligamentum Flavum Hypertrophy Through the LPAR1/Akt Pathway

黄韧带肥厚病理机制

信号通路靶向治疗

Background/Aims: Hypertrophic ligamentum flavum (LF) is a major cause of lumbar spinal stenosis. Our previous work showed that high levels of lysophosphatidic acid (LPA) expression are positively corr

Figshare2018-02-21 更新50

GSE131907

癌症基因组数据

肺腺癌转移机制

11 tumour, 11 distant normal lung, 10 normal lymph node, and 10 metastatic brain tissue samples were obtained from LUAD patients conserving surgery without prior treatment. 7 metastatic lymph node and

Figshare2022-05-11 更新50

Identification of Metastasis-prone Lung Adenocarcinoma Cell Population That Is Sensitive to Notch Inhibition

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