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Noncoding RNA expression profile of dasatinib-treated chronic myeloid leukemia patients with resistance to imatinib

ChEBI2013-07-31 更新2026-07-21 收录

白血病耐药机制

非编码RNA表达谱

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-33288 数据链接链接失效反馈

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资源简介：

Noncoding RNA expression profile of dasatinib-treated chronic myeloid leukemia patients with resistance to imatinib

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创建时间：

2013-07-31

白血病耐药机制

RNAm6A修饰分析

N6-Methyladenosine (m6A) is the most abundant post-transcriptional modification in eukaryotes, the imbalance of which is reported to be associated with various pathological processes, including drug r

NIAID Data Ecosystem60

Additional file 3 of Small nucleolar RNA dysregulation and potential roles in bovine subclinical mastitis

乳腺炎分子病理

非编码RNA表达谱

Supplementary Material 3. Table S3A List of differntially expressed snoRNAs in Staphylococcus aureus versus control group. Table S3B List of differntially expressed snoRNAs in Staphylococcus chromogen

Figshare2025-09-12 更新30

Mus musculus Raw sequence reads. Mus musculus

脓毒症急性器官损伤分子机制

非编码RNA表达谱

Systematic analysis of the kidney expression profile of non-coding RNAs involved in ischemia/reperfusion-induced acute kidney injury in mice using RNA sequencing

NIAID Data Ecosystem30

Data_Sheet_1_Acute Myeloid Leukemia Chemo-Resistance Is Mediated by E-selectin Receptor CD162 in Bone Marrow Niches.PDF

白血病耐药机制

肿瘤微环境靶向治疗

The interactions of leukemia cells with the bone marrow (BM) microenvironment is critical for disease progression and resistance to treatment. We have recently found that the vascular adhesion molecul

NIAID Data Ecosystem40

Coding and noncoding gene expression of 48 pediatric AML samples. Homo sapiens

儿童急性髓系白血病分子特征

非编码RNA表达谱

Long non-coding RNAs (lncRNAs) and miRNAs have emerged as crucial regulators of gene expression and cell fate decisions. Here we present an integrated analysis of the ncRNA-transcriptome of purified h

NIAID Data Ecosystem30

Noncoding RNA expression profile of dasatinib-treated chronic myeloid leukemia patients with resistance to imatinib

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