Low KLF11 Determines the CSCâlike Identity in Sarcoma via restraining YAP transactivity
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https://www.ncbi.nlm.nih.gov/sra/SRP211800
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资源简介：
We conducted transcriptome comparison of control and KLF11-knockdown RPKS1 cells, then ChIP-seq experiments to investigate the role of KLF11 in YAP/TEAD transcriptional activity, and Transposase-accessible chromatin using sequencing (ATAC-seq) experiments to compare chromatin open regions between control and KLF11-knockdown cells.
创建时间：
2020-06-26
