Diverse mitochondrial abnormalities in a new cellular model of TAFFAZZIN deficiency are remediated by Cardiolipin-interacting small molecules
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https://www.omicsdi.org/dataset/pride/PXD026731
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资源简介：
With the goal of identifying unappreciated pathways of mitochondrial dysregulation in Barth Syndrome, we utilized an unbiased proteomics strategy to identify pathways of metabolic dysregulation in an HEK293-based TAZ-deficiency model.
创建时间：
2022-02-17
