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SLIT2 overexpression in periodontitis intensifies inflammation and alveolar bone loss.

NIAID Data Ecosystem2026-03-11 收录

牙周炎分子机制

Slit2

数据链接：

https://www.ncbi.nlm.nih.gov/sra/SRP267596 数据链接链接失效反馈

官方服务：

资源简介：

For better evaluation of the role of Slit2 in the development of periodontitis.

应用场景：

创建时间：

2020-06-18

牙周炎分子机制

免疫原性细胞死亡

BackgroundRecent studies have suggested that cell death may be involved in bone loss or the resolution of inflammation in periodontitis. Immunogenic cell death (ICD), a recently identified cell death

NIAID Data Ecosystem40

Redondoviridae infection regulates circRNAome in periodontitis. Redondoviridae infection regulates circRNAome in periodontitis

病毒-宿主相互作用

牙周炎分子机制

Redondoviridae is a recently identified family of DNA viruses associated with periodontitis. Circular RNAs (circRNAs) emerged as a novel endogenous, conserved noncoding RNAs, which contributed to the

NIAID Data Ecosystem20

Additional file 2 of Comprehensive analysis of DNA methylation for periodontitis

牙周炎分子机制

DNA甲基化分析

Additional file 2. Gene ontology results.

Figshare2024-02-15 更新20

C5aR antagonist inhibits LPS-induced inflammation in human gingival fibroblasts via NF-κB and MAPK signaling pathways

牙周炎分子机制

抗炎药物药理学

Objective: Abnormal complement activation is associated with periodontitis. W54011 is a novel non-peptide C5aR antagonist (C5aRA) that exhibits favorable anti-inflammatory effects in various inflammat

DataCite Commons2025-07-10 更新30

Additional file 8 of Identification and assessment of differentially expressed necroptosis long non-coding RNAs associated with periodontitis in human

牙周炎分子机制

lncRNA诊断标志物

Additional file 8. The ROC sensitivities of 10-lncRNAs in GSE16134 dataset.

Figshare2024-08-13 更新20