small RNA from N-TERA cells with different treatments. Homo sapiens

NIAID Data Ecosystem2026-03-09 收录

small RNA表达分析

细胞分化调控

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA301447 数据链接链接失效反馈

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资源简介：

Detect the implication of the Dioxin Receptor in the differentiation of N-TERA cells after Retinoic Acid treatment

创建时间：

2015-11-06

Gene expression comparisons of megakaryocyte-erythrocyte progenitors isolated from wild type and c-MybTG/c-MybTG mice

细胞分化调控

转录因子功能研究

Transgene insertion in proximity to the c-Myb gene disrupts c-Myb expression in megakaryocyte-erythrocyte progenitors (MEPs), resulting in skewed commitment to megakaryocyte lineage. To understand the

NIAID Data Ecosystem50

A Transcription Factor Atlas of Directed Differentiation [10XscRNA_180124_perturb]

细胞分化调控

单细胞转录组学

10X single-cell RNA-seq data profiling hESCs overexpressing transcription factors Overall design: hESCs were transduced with a library of 90 transcription factor ORFs and cultured for 7 days before si

NIAID Data Ecosystem10

STAT4 increases the phenotypic and functional heterogeneity of tissue-resident memory T cells

细胞分化调控

单细胞转录组学

We identified STAT4 as a regulator of tissue-resident memory T cell differentiation after infection. Single cell RNA sequencing was used to define the heterogeneity in gene expression among T cells in

NIAID Data Ecosystem00

The kinase DYRK1A reciprocally regulates the differentiation of Th17 and regulatory T cells [seq]

细胞分化调控

结肠炎机制

Using an unbiased chemical biology approach, we discover harmine as a novel regulator of Treg/Th17 differentiation. Harmine enhances Treg differentiation (working in conjunction with low levels of exo

NIAID Data Ecosystem20

Regulation of Tfh differentiation by Mettl3 [RNA-seq]

细胞分化调控

表观转录组学

T follicular helper (Tfh) cells are a specialized subset of CD4 T cells that are essential for most antibody productions and humoral responses. It is well characterized that transcriptional network ex

NIAID Data Ecosystem10

small RNA from N-TERA cells with different treatments. Homo sapiens

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