Additional file 1 of Multi-omics of the esophageal microenvironment identifies signatures associated with progression of Barrett’s esophagus

Additional file 1: Table S1. Transcripts differentially expressed between normal and GERD samples. Table S2. Transcripts differentially expressed between normal and MET samples. Table S3. Pathway analysis of differentially expressed transcripts. Table S4. Genes identified with possible differences in splicing events. Table S5. Intron retention and exon skipping events found between disease groups. Table S6. Circular RNAs tested for differential prevalence within disease groups. Table S7. Gene fusion events identified within the esophageal samples. Table S8. Per taxa differences between saliva and esophageal samples. Table S9. Per taxa differences between normal and GERD esophageal samples. Table S10. Per taxa differences between normal and MET esophageal samples. Table S11. Per taxa differences between normal and disease esophageal samples. Table S12. MINE analysis between Campylobacter and transcripts. Table S13. MINE analysis between Campylobacter iOTU36 and transcripts. Table S14. Conservation of proteins unique to GS1 C. concisus isolates. Table S15. Proteins unique to C. concisus ESOS44-1. Table S16. Differential expression between control and Campylobacter ESOS13-1. Table S17. Differential expression between control and Campylobacter ESOS14-1. Table S18. Differential expression between control and Campylobacter ESOS18-1. Table S19. Differential expression between control and Campylobacter ESOS33-1. Table S20. Differential expression between control and Campylobacter ESOS44-1. Table S21. Differential expression between control and Campylobacter ESOS44-4.