Transcription profiling of human CML CD34+ cells during Imatinib therapy

ChEBI2008-10-21 更新2026-07-21 收录

慢性髓性白血病

干细胞转录组学

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-12211 数据链接链接失效反馈

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Transcription profiling of human CML CD34+ cells during Imatinib therapy

创建时间：

2008-10-21

Downregulation of Smad4 expression confers chemoresistance against imatinib mesylate to chronic myeloid leukemia K562 cells

慢性髓性白血病

药物耐药性

Objective: Imatinib mesylate (IM), a tyrosine kinase inhibitor, exhibits clinically prominent effects against chronic myeloid leukemia (CML); however, a few patients have shown resistance to IM

DataCite Commons2024-02-20 更新60

RNA-Seq of WT and UBQLN1 KO hESCs treatment with 5%O2 or NAC. RNA-Seq of WT and UBQLN1 KO hESCs treatment with 5%O2 or NAC

干细胞转录组学

基因表达调控

We aimed to investigate the transcriptional change after treatment with NAC or 5% O2 Overall design: The Wild type and UBQLN1-/- hESCs treatment with NAC or 5% O2

NIAID Data Ecosystem30

A Tumor Suppressor Function of LncRNA-X in Bcr-Abl-induced Tumorigenesis

慢性髓性白血病

长链非编码RNA抑癌功能

Aberrant long noncoding RNA (lncRNA) expression has been described in many human malignancies, including leukemia. Philadelphia-positive (Ph+) chronic myeloid leukemia (CML) is a stem cell disease ind

NIAID Data Ecosystem10

Transcriptome analysis of Human Trophoblast Stem cell (HTS) and TEAD4 knock down HTS cells by next generation sequencing

干细胞转录组学

TEAD4基因功能

mRNA profiles of control Human Trophoblast Stem cell (HTS) and TEAD4 knock down HTS cells were generated by deep sequencing, in triplicate, using Illumina NovaSeq 6000 platform. TEAD4 Knock down in HT

NIAID Data Ecosystem50

Retrograde movements determine effective stem cell numbers in intestinal crypts

干细胞转录组学

隐窝细胞异质性分析

Our goal was to compare the transcriptomic profile of lgr5+ high, medium and low cells (corresponding to centre, border and >3 row in crypts) between small and large intestine in mice. We observed dif

NIAID Data Ecosystem20

Transcription profiling of human CML CD34+ cells during Imatinib therapy

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