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Gene expression patterns in developing intestinal organoids exposed to prolonged FGF4- and Wnt-signaling

ChEBI2016-07-04 更新2026-07-12 收录

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基因信号通路

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-4168 数据链接链接失效反馈

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Gene expression patterns in developing intestinal organoids exposed to prolonged FGF4- and Wnt-signaling

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创建时间：

2016-07-04

单细胞转录组学

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High-throughput single-cell RNA-sequencing (scRNA-seq) has transformed our ability to resolve cellular properties across systems. A key scRNA-seq catalyzer was the introduction of microdroplet-based s

NIAID Data Ecosystem60

Murine intestinal organoid scEU-seq data - Raw

单细胞转录组学

肠道类器官

AnnData object of scRNA-seq dataset of murine intestinal organoids metabolically labeled and sequenced with scEU-seq. The data has been collected as provided by the original publication in an AnnData

Figshare2023-07-24 更新40

C12orf43-NAT10 Enhance breast cancer proliferation and chemotherapy resistance via BMI1-P53 signaling axis in a N4-acetylcytidine-dependent manner

化疗耐药性

基因信号通路

C12orf43-NAT10 interaction was found to accelerate breast cancer progression and chemotherapy resistance by activating the BMI-P53 signaling axis in a N4-acetylcytidine-dependent manner. 1.MCF7/ADR_vs

NIAID Data Ecosystem20

Generation of intestinal organoids from human urine cells. Generation of intestinal organoids from human urine cells

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尿液细胞重编程

Intestinal organoids are ideal models for studying human intestinal diseases. We report functional intestinal organoid models (U-iIOs) generated by reprogramming human urinary epithelial cells (hUCs).

NIAID Data Ecosystem50

Single cell sequencing of human intestinal organoid cells.. Single cell sequencing of human intestinal organoid cells.

单细胞RNA测序

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Unbiased and FACS was performed on human intestinal organoid cells and coupled to single cell sorting by the SortSeq protocol (Muraro et al., 2016). Overall design: Intestinal organoid differentiation

NIAID Data Ecosystem30

Gene expression patterns in developing intestinal organoids exposed to prolonged FGF4- and Wnt-signaling

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