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Capturing the biological impact of the melanoma susceptibility genes CDKN2A and MC1R in cocultured keratinocytes and melanocytes

ChEBI2013-12-31 更新2026-07-21 收录

黑色素瘤分子机制

细胞共培养模型

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-44805 数据链接链接失效反馈

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资源简介：

Capturing the biological impact of the melanoma susceptibility genes CDKN2A and MC1R in cocultured keratinocytes and melanocytes

应用场景：

创建时间：

2013-12-31

黑色素瘤分子机制

基因功能注释

Significant GO terms for the top 300 genes that distinguish primary from cutaneous melanoma.

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Importance of RAD6B in activation of canonical WNT signaling in melanoma development

黑色素瘤分子机制

Wnt信号通路调控

We have demonstrated that RAD6B, a ubiquitin conjugating enzyme that is a principal component of translesion synthesis pathway, stabilizes and transcriptionally activates beta-catenin by lysine 63-lin

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Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance. Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

黑色素瘤分子机制

自噬调控

To Study the role of mutant Braf V600E and ERK dependent phosphorylation of TFEB in regulation of autophagy and lysosome biogenesis in the context of melanoma growth and metastasis, we used whole gene

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Permeability_IMR90-4_EECM-BMEC-BPLC-Cocultures_RawFiles_NotForPublication

细胞渗透性研究

细胞共培养模型

Raw data excel files for permeability studies on EECM-BMEC-like cell and BPLC cocultures, which were not included in publication as they only contain non-stimulated data or were excluded for other sta

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m6A IP sequencing analysis of m6A mRNA methylation regulation by FTO in melanoma cells. m6A IP sequencing analysis of m6A mRNA methylation regulation by FTO in melanoma cells

RNA表观遗传学

黑色素瘤分子机制

N6-methyladenosine (m6A) RNA methylation is the most abundant internal chemical modifications in eukaryotic messenger RNA (mRNA) as well as long non-coding RNA (lncRNA). Recently, m6A RNA methylation

NIAID Data Ecosystem50

Capturing the biological impact of the melanoma susceptibility genes CDKN2A and MC1R in cocultured keratinocytes and melanocytes

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