首页/数据集/The gene set related to the “Cytotoxicity” and “Progenitor exhaustion” function of T cells.

The gene set related to the “Cytotoxicity” and “Progenitor exhaustion” function of T cells.

Figshare2025-11-04 更新2026-04-28 收录

数据链接：

https://figshare.com/articles/dataset/The_gene_set_related_to_the_Cytotoxicity_and_Progenitor_exhaustion_function_of_T_cells_/30534406 数据链接链接失效反馈

官方服务：

资源简介：

The gene set related to the “Cytotoxicity” and “Progenitor exhaustion” function of T cells.

应用场景：

创建时间：

2025-11-04

相关数据集

BM21 Tumor

肿瘤免疫

CD8T细胞分化

The purpose of this experiment is to follow the differentiation of naive CD8 T cells in memory CD8 T cells in response tumor growth. To do so, naive C57Bl/6J mice were transferred with naive F5 TCR-tr

NIAID Data Ecosystem90

scRNA-seq of antigen-specific CD8+ T cells from tumor draining LN, TIL and PBMC of melanoma bearing mice

肿瘤免疫

T细胞耗竭

CD8+ T cell-mediated immune response plays a pivotal role in controlling tumor growth.However, within the tumor microenvironment (TME), prolonged antigen exposure, as well as immunosuppressive factors

NIAID Data Ecosystem60

Pro-survival Lipid Sphingosine-1-Phosphate Metabolically Reprograms T cell to Limit its Anti-Tumor Activity

免疫代谢

T细胞功能调控

Sphingosine 1-phosphate (S1P), a pro-tumor survival bioactive lysophospholipid, generated by sphingosine kinase 1 (SphK1), regulates lymphocyte egress into circulation via transducing S1P receptor 1 (

NIAID Data Ecosystem50

Induction of antitumor activity of CD8+ T cells by inactivation of ATPIF1 through F1Fo-ATPase-mediated metabolic reprogramming. Induction of antitumor activity of CD8+ T cells by inactivation of ATPIF1 through F1Fo-ATPase-mediated metabolic reprogramming

肿瘤免疫

单细胞测序

In this study, CD45+ leukocytes were sorted from the mixed tumor samples, which were collected from the C57BL/6 wild type mice and ATPIF1 knock-out mice, respectively. The live CD45+ cells were used f

NIAID Data Ecosystem60

Additional file 7: Table S7. of Metronomic cyclophosphamide activation of anti-tumor immunity: tumor model, mouse host, and drug schedule dependence of gene responses and their upstream regulators

基因表达

肿瘤免疫

Genes whose basal expression in untreated GL261(B6) tumors is either higher or lower than in untreated LLC and untreated B16F10 tumors. (XLSX 670 kb)

DataCite Commons2024-12-17 更新60