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Killer cell Ig-like receptor (KIR) 3DL1 downregulation enhances inhibition of type 1 diabetes by autoantigen-specific regulatory T cells

ChEBI2011-01-06 更新2026-07-21 收录

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-26456 数据链接链接失效反馈

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Killer cell Ig-like receptor (KIR) 3DL1 downregulation enhances inhibition of type 1 diabetes by autoantigen-specific regulatory T cells

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2011-01-06

自身免疫疾病免疫治疗

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Progression towards type 1 diabetes (T1D) in susceptible patients is linked to a progressive decline in the capacity of regulatory T cells (Treg) to maintain tolerance. As such, therapies aimed at red

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Butyrate-modified liposomes target the delivery of itaconic acid to modulate the Th17/Treg cell balance through epigenetic reprogramming, improving primary biliary cholangitis

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Primary biliary cholangitis (PBC) is a chronic autoimmune disease. This study aims to design itaconic acid (IA)-loaded butyrate (Bu)-modified lipid nanoparticles (LNP-Bu@IA) for targeted delivery of I

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Killer cell Ig-like receptor (KIR) 3DL1 downregulation enhances inhibition of type 1 diabetes by autoantigen-specific regulatory T cells

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