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2024-05-13
相关数据集
Distinct sub-cellular autophagy impairments independent of protein aggregation in induced neurons from patients with Huntington disease. Distinct sub-cellular autophagy impairments independent of protein aggregation in induced neurons from patients with Huntington disease
Huntington’s disease(HD)is a hereditary neurodegenerative disorder that is caused by CAGexpansionsin the huntingtin(HTT)gene. Modelling HD in the lab has proven challenging asrodent models poorly repr
NIAID Data Ecosystem70
High-resolution Sp1 DNA-binding Site Mapping by ChIP-exo in STHdh Q7/Q7 cells
High-resolution Sp1 DNA-binding Site Mapping by ChIP-exo in STHdh Q7/Q7 cells
ChEBI2016-07-15 更新40
Single-nucleus RNA-sequencing of unaffected control and Huntington's disease GLAST-positive iPSC-derived astrocytes. Single-nucleus RNA-sequencing of unaffected control and Huntington's disease GLAST-positive iPSC-derived astrocytes
Huntington's disease (HD) and control GLAST-postive induced pluripotent stem cell (iPSC)-derived astrocytes underwent single-nucleus RNA-sequencing to investigate cell state diversity across control a
NIAID Data Ecosystem10
Euploid HD iPSC lines (e.g. lines with a normal karyotype when first generated) accumulate abnormalities at the same rate as euploid control lines.
Euploid HD iPSC lines (e.g. lines with a normal karyotype when first generated) accumulate abnormalities at the same rate as euploid control lines.
NIAID Data Ecosystem50
Structural and functional features of medium spiny neurons in the BACHDÎN17 mouse model of Huntingtonâs disease
In the BACHD mouse model of Huntingtonâs disease (HD), deletion of the N17 domain of the Huntingtin gene (BACHDÎN17, Q97) has been reported to lead to nuclear accumulation of mHTT and exacerbation
DataONE2024-07-18 更新30
