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reveal the regulation mechanism of DPYSL2 on AML
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创建时间：
2025-02-04
相关数据集
Additional file 6 of The landscape of gene co-expression modules correlating with prognostic genetic abnormalities in AML
Additional file 6: Table S2. The overlapped genes of modules correlating with NPM1 significantly, by analysis based on TCGA and BeatAML database, respectively.
NIAID Data Ecosystem70
Contrasting the co-occurrence patterns of NPM1-FLT3-ITD and NPM1-RAS mouse acute myeloid leukemias by whole exome sequencing.. Molecular Synergy in NPM1 AML
Mutations affecting NPM1 define the commonest subgroup of acute myeloid leukemia (AML). They frequently co-occur with mutations of FLT3, usually internal tandem duplications (ITD), and less commonly o
NIAID Data Ecosystem50
Supplementary Table 10 from Disruption of the MYC Superenhancer Complex by Dual Targeting of FLT3 and LSD1 in Acute Myeloid Leukemia
Correlation of Regulon Enrichment Signatures with MYC Gene Expression in Primary AML
NIAID Data Ecosystem40
35549e9c-f1f9-4722-8181-ec8c3cd8fbbf - samples
Whole genome sequencing of AML blood or bone marrow at presentation and remission for 5 patients. Relapse samples are included for 2 patients, totaling 12 WGS BAM files.EGA dataset EGAD00001005120
NIAID Data Ecosystem70
Isoform- and mutation- specific roles of Wilms Tumor 1 in acute myeloid leukemia maintenance (DNAse I hypersensitivity). Isoform- and mutation- specific roles of Wilms Tumor 1 in acute myeloid leukemia maintenance (DNAse I hypersensitivity)
Acute myeloid leukemia (AML) is caused by recurrent mutations in members of the gene regulatory and signalling machinery subverting hematopoietic differentiation. We previously showed that each AML su
NIAID Data Ecosystem40
