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Effects of bisphenol A on gene expression in adipocytes from lean children: association to metabolic disorders

ChEBI2015-07-09 更新2026-07-12 收录

内分泌干扰物毒性

儿童代谢疾病机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-58516 数据链接链接失效反馈

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Effects of bisphenol A on gene expression in adipocytes from lean children: association to metabolic disorders

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创建时间：

2015-07-09

内分泌干扰物毒性

胰腺β细胞分化机制

Bisphenol-A is a widespread endocrine disruptor chemical. In utero or perinatal exposure to bisphenol-A (BPA), leads to impaired glucose metabolism during adulthood. To investigate the consequences o

NIAID Data Ecosystem50

Table_2_Exposure to Dibutyl Phthalate and Reproductive-Related Outcomes in Animal Models: Evidence From Rodents Study.docx

内分泌干扰物毒性

动物实验meta分析

Background: Dibutyl phthalate (DBP) was an endocrine disruptor, which may lead to cancer and affects reproductive function when accumulated in the body. But the precise role of DBP in the reproductive

NIAID Data Ecosystem50

Potential mechanisms of endocrine disruptors as revealed by uterine gene profile

内分泌干扰物毒性

生殖系统转录组

Potential mechanisms of endocrine disruptors as revealed by uterine gene profile

ChEBI2010-10-06 更新20

Exposure to polybrominated biphenyl (PBB) associates with DNA methylation differences across the genome. Exposure to polybrominated biphenyl (PBB) associates with DNA methylation differences across the genome

环境基因组学

内分泌干扰物毒性

Polybrominated biphenyl (PBB) is an endocrine-disrupting compound which was accidently added to the food supply in Michigan in the 1970’s. Highly exposed individuals and their children have numerous h

NIAID Data Ecosystem50

Effect of maternal exposure to dioxin on the pituitary and hypothalamic expression of genes in PND70 male pups. Rattus norvegicus

内分泌干扰物毒性

神经内分泌基因调控

Our previous studies have revealed that treatment of pregnant rats with 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD, 1 μg/kg) at gestational day (GD) 15 reduces the pituitary synthesis of luteinizing ho

NIAID Data Ecosystem40

Effects of bisphenol A on gene expression in adipocytes from lean children: association to metabolic disorders

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