Minimal Invariant Mitochondrial Kernel, Rank-Control, Dual-Anchor Validation, and TOP3A-RMI2 Blind-Spot Resolution in a Computational Longevity Discovery Pipeline (Trinity Claims #36-#39, 2026-03-17)

This version updates the previously published Zenodo record for Trinity Claim #36 with three additional results, Trinity Claims #37, #38, and #39. Claim #37: full mitochondrial kernel retention is not explained only by top-rank position. Claim #38: the invariant mitochondrial kernel compresses around two duplicated anchor genes, MRPL9 and ATP5F1E. Claim #39: a masked DNA blind spot resolves into a TOP3A-RMI2 checkpoint microkernel. Phase-12 blind-spot synthesis ranks the two TOP3A-RMI2 triads at #1 and #4 among masked candidates, and phase-16 retained-kernel compression shows empirical p=0.000840 in the rank-bounded null and p=0.000620 in the full cb_1_g_small null. Core retained signal remains:- mito 7/7- immune 4/6- DNA 3/4- cilia 2/3 This version contains Claims #36-#39, phase-12 blind-spot synthesis, phase-13 retention audit, phase-14 rank-artifact control, phase-15 anchor-compression analysis, phase-16 theme-kernel compression analysis, and the updated Physiology or Medicine summary. This is a computational discovery package and not a completed causal validation study.